n.激肽释放酶

Kallikrein is an enzyme that plays a crucial role in the regulation of blood pressure and inflammation.
噬菌体是一种在血压调节和炎症反应中起着关键作用的酶。
The kallikrein-kinin system is activated during tissue injury, leading to vasodilation and increased permeability of blood vessels.
在组织损伤过程中，激肽释放酶-激肽系统被激活，导致血管扩张和血管通透性增加。
Increased urinary kallikrein levels may be an indicator of kidney damage or disease.
尿液中激肽释放酶水平升高可能是肾脏损害或疾病的一个标志。
Kallikrein cleaves kininogen to release bradykinin, which is a potent vasodilator.
激肽释放酶裂解激肽原以释放缓激肽，这是一种强效的血管舒张剂。
Several studies have suggested that kallikrein inhibitors could be potential therapeutic targets for hypertension treatment.
多项研究表明，激肽释放酶抑制剂可能是高血压治疗的潜在靶点。
Mutation in the kallikrein gene has been associated with hereditary angioedema.
激肽释放酶基因突变与遗传性血管性水肿有关。
Intravenous administration of kallikrein can help reduce blood viscosity and improve microcirculation.
静脉注射激肽释放酶有助于降低血液粘稠度并改善微循环。
Some snake venoms contain kallikrein-like enzymes that can trigger similar physiological effects as endogenous kallikreins.
一些蛇毒含有类似于内源性激肽释放酶的酶，可以引发相似的生理效应。
Kallikrein-8, also known as prostate-specific antigen (PSA), is widely used as a biomarker for prostate cancer screening.
激肽释放酶-8，也称为前列腺特异性抗原（PSA），被广泛用于前列腺癌筛查的生物标志物。
Research on the kallikrein-kinin system has expanded our understanding of pain modulation and inflammatory processes.
对激肽释放酶-激肽系统的研究扩展了我们对疼痛调控和炎症过程的理解。
请注意：以上中文解释基于原文直译，部分内容可能需要结合医学背景知识理解。