微自噬

Microautophagy involves the selective degradation of cytoplasmic components by autophagosomes within the lysosome.
这意味着微自噬通过溶酶体内的自噬体选择性地降解细胞质成分。
In neurons, microautophagy is a primary mechanism for removing damaged organelles and proteins.
在神经元中，微自噬是清除受损细胞器和蛋白质的主要途径。
Mitochondria are frequently targeted for microautophagy during cellular stress conditions.
细胞压力条件下，线粒体常常成为微自噬的目标。
Autophagy researchers have observed microautophagy in various cell types, from yeast to humans.
自噬研究者已经在从酵母到人类的多种细胞类型中观察到了微自噬。
Dysregulation of microautophagy can lead to neurodegenerative diseases like Alzheimer's.
微自噬的失调可能导致如阿尔茨海默病之类的神经退行性疾病。
The process of microautophagy requires the formation of a double-membrane vesicle called an amphisome.
微自噬过程需要形成一个名为“amphisome”的双层膜囊泡。
Researchers found that aging cells exhibit increased levels of microautophagy to maintain cellular homeostasis.
研究人员发现，随着细胞衰老，微自噬水平增加以维持细胞稳态。
In cancer cells, microautophagy may contribute to resistance to chemotherapy drugs.
在癌细胞中，微自噬可能有助于抵抗化疗药物的作用。
Proteins involved in microautophagy often have specific domains that facilitate their recognition and engulfment by autophagosomes.
参与微自噬的蛋白通常具有特定区域，帮助它们被自噬体识别并吞噬。
Further studies are needed to fully understand the role of microautophagy in cellular health and disease.
进一步的研究尚需深入理解微自噬在细胞健康和疾病中的作用。