n.神经病发病机理

Neuropathogenesis refers to the molecular and cellular mechanisms that lead to nerve damage or dysfunction.
神经病理学是指导致神经损伤或功能障碍的分子和细胞机制。
The study of neuropathogenesis has elucidated the role of inflammation in demyelination.
研究神经病理学揭示了炎症在脱髓鞘中的作用。
Autoimmune diseases often exhibit a complex neuropathogenesis involving both genetic and environmental factors.
自身免疫性疾病通常涉及遗传和环境因素的复杂神经病理学过程。
In Parkinson's disease, the primary neuropathogenesis is the loss of dopaminergic neurons.
在帕金森病中，主要的神经病理学过程是多巴胺能神经元的丧失。
Alcohol abuse can lead to axonal injury, a key component of neuropathogenesis.
酒精滥用会导致轴突损伤，这是神经病理学的重要环节。
The spread of neurotoxins during a viral infection can cause direct neuropathogenesis.
病毒性感染期间神经毒素的传播可直接引起神经病理学改变。
Genetic mutations can initiate neuropathogenesis in inherited disorders like Charcot-Marie-Tooth disease.
遗传突变在如Charcot-Marie-Tooth病等遗传性疾病中可以启动神经病理学进程。
Oxidative stress plays a significant role in the pathogenesis of several neurodegenerative diseases.
氧化应激在多种神经退行性疾病的发生中起着重要作用。
Neuropathogenesis of diabetes mellitus involves damage to blood vessels and nerves.
糖尿病的神经病理学过程涉及血管和神经的损伤。
Understanding neuropathogenesis can pave the way for effective treatments and preventive measures.
理解神经病理学有助于开发有效的治疗策略和预防措施。