The neurotransmitter acetylcholine is released from the
presynaptic terminal and binds to receptors on the postsynaptic neuron.
神经递质乙酰胆碱从突触前末梢释放,并与突触后神经元上的受体结合。
In a synapse, the
presynaptic neuron's axon terminal releases chemical messengers called neurotransmitters.
在突触中,轴突末端的突触前神经元释放化学信使,即神经递质。
The stimulation of a sensory receptor initiates an electrical signal that travels along the axon to the
presynaptic terminal.
感官受体的刺激引发电信号沿轴突传至突触前末梢。
The
presynaptic vesicles contain neurotransmitters that are stored and ready to be released upon receiving an action potential.
突触前囊泡内储存着神经递质,一旦接收到动作电位就会释放出来。
The
presynaptic membrane contains voltage-gated calcium channels that allow calcium ions to enter during an action potential.
突触前膜包含电压门控钙通道,使钙离子在动作电位期间进入。
The release of dopamine from the
presynaptic terminal is essential for reward-motivated behavior in the brain.
脑中来自突触前末梢的多巴胺释放对于奖赏驱动的行为至关重要。
Presynaptic inhibition can modulate the strength of synaptic transmission by blocking neurotransmitter release at the synapse.
突触前抑制通过阻止神经递质在突触处的释放,调节突触传递的强度。
Long-term potentiation (LTP) at the synapse occurs when repeated activation of the
presynaptic neuron enhances the postsynaptic response.
长期增强(LTP)在突触发生时,当突触前神经元被反复激活时,会增强突触后响应。
Neurotransmitters like serotonin and norepinephrine are synthesized in the
presynaptic neurons and then transported across the synaptic cleft.
例如血清素和去甲肾上腺素这样的神经递质在突触前神经元中合成,然后跨过突触间隙运输。
Damage to the
presynaptic terminal can lead to a decrease in neurotransmission, contributing to symptoms of neurological disorders.
突触前末梢的损伤可能导致神经递质传递减少,从而引发神经系统疾病的症状。
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