Diacylglycerol is a key intermediate in the process of cellular respiration.
这是一个关键的中间产物在细胞呼吸过程中。
The activation of protein kinase C by
diacylglycerol leads to changes in cell signaling pathways.
二酰基甘油通过激活蛋白激酶C引发细胞信号传导途径的变化。
Endocannabinoids, like anandamide, can increase intracellular
diacylglycerol levels, leading to enhanced appetite.
内源性大麻素如安非他命可以提高细胞内的二酰基甘油水平,从而增强食欲。
Activation of phospholipase C by
diacylglycerol generates inositol trisphosphate and
diacylglycerol.
二酰基甘油通过磷脂酶C的激活产生肌醇三磷酸和二酰基甘油。
Diacylglycerol serves as a second messenger in the regulation of lipid metabolism in cells.
二酰基甘油作为细胞中脂质代谢的第二信使。
Cells respond to stress by increasing
diacylglycerol production, which activates stress response pathways.
细胞在应激状态下通过增加二酰基甘油的生成来激活应激反应途径。
Diacylglycerol lipases play a crucial role in breaking down this molecule back into fatty acids and glycerol.
二酰基甘油酯酶在分解该分子回脂肪酸和甘油中起着关键作用。
In adipose tissue,
diacylglycerol is an important precursor for the synthesis of triglycerides.
在脂肪组织中,二酰基甘油是合成甘油三酯的重要前体。
The elevation of
diacylglycerol levels can lead to the closure of calcium channels in cells, affecting cell contraction.
二酰基甘油水平的升高可能导致细胞内钙通道关闭,影响细胞收缩。
Research suggests that certain drugs target
diacylglycerol signaling pathways for therapeutic purposes.
研究表明,某些药物通过靶向二酰基甘油信号通路具有治疗目的。
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